Intentional Genetic Creation of Cancer in the COVID-19 Vaccines with SV40. Robert Malone should be able to help us understand "Turbo Cancer" much better, I think.

SV40, UCSF, Salk Insitute

A disturbing history of research involving Robert Malone reveals that he should know a lot more about “Turbo Cancers” caused by COVID-19 vaccines than he has revealed to the public.

This article brings forward some information from the 1950s - 1980s, then skips forward to the present. It’s by no means complete, but gives an outline that involves Robert Malone. As always with this type of sinister research, so terrible that it defies description, there were many people involved, as was the case with mRNA “vaccine” technology for which the Nobel Prize spurned Robert Malone by awarding the 2023 Nobel Prize in Physiology or Medicine jointly to Katalin Karikó and Drew Weissman for their contributions to the global mass murder program. (Robert Malone had been in touch with people familiar with the Nobel Prize Committee process going back to at least 2021. I doubt he wanted the Nobel Prize under the circumstances, although he deserves it, especially now in light of the inclusion of SV40 DNA in the COVID-19 vaccines).

The difference between most of the evil people in the BioDefense/Countermeasure industry and Robert Malone is that Malone presents himself as an insider/whistleblower. However a close examination of Malone’s background raises the strong possibiltiy that he is hiding some of the most important information about his research from the public.

The inclusion of SV40 DNA sequence in the COVID-19 vaccines appears to be an effort to make humans express genes that will pass along cancer-producing genetics to their offspring over time. This has already occurred on a massive scale, as parasites on the human race like Elon Musk, Pfizer and Moderna pursue “mRNA Cancer Vaccines,” knowing full well what is to come and is already under way. (1).

The following screenshot was taken from a slideshow presentation by Tiffany Seagroves of UC-Davis on the “Mammary Mouse Tumor Model” (2012). More on this model later. The Mouse Mammary Tumor Model was a cancer-producing effot that Robert Malone was “deeply” involved with.

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1955-1963. SV40, Simian Virus, is included in the Salk Polio vaccines, creating massive amounts of cancer today. (Polio was not caused by a virus in the first place, but instead was caused by Lead, Arsenic, and DDT.

The literature surrounding this intentional creation of cancer with the inclusion of SV40 by Jonas Salk in the polio vaccines is still referred to as a “contamination,” not something intentional, even though they’ve done it again on a truly sad and massive scale. Robert Malone learned his trade at the Salk Institute.

Immunization Safety Review: SV40 Contamination of Polio Vaccine and Cancer, 2002, provides a good read, while also using the disinformation term “contamination.”

The discovery of the polyomavirus SV40, as well as its introduction as a pathogen into the human population, was tied to the development and worldwide distribution of early forms of the polio vaccine (13, 95, 111, 123). Inactivated (Salk) and early live attenuated (Sabin) forms of polio vaccines were inadvertently contaminated with SV40 (95, 97, 111). In addition, different adenovirus vaccines distributed to some U.S. military personnel from 1961 to 1965 also contained SV40 (64). The viral contamination occurred because these early vaccines were prepared in primary cultures of kidney cells derived from rhesus monkeys, which are often naturally infected with SV40 (13, 95, 111). Infectious SV40 survived the vaccine inactivation treatments, and conservative estimates indicate that up to 30 million people (children and adults) in the United States may have been exposed to live SV40 from 1955 through 1963 when administered potentially contaminated polio vaccines (95, 111). Millions of people worldwide were also potentially exposed to SV40 because contaminated polio vaccines were distributed and used in many countries (85, 123). These data led the Institute of Medicine to conclude that “the biological evidence is of moderate strength that SV40 exposure from the polio vaccine is related to SV40 infection in humans” (111).

1975

Efforts to develop genes that create cancer in mammals are initimately associated with the Salk Institute and the University of California. In 1975, future Nobel Prize winners J. Michael Bishop and Harold E. Varmus of UC-San Fransisco and Salk Institute “demonstrated the true origins of oncogenes,” according to text of the Nobel Prize committee.

Etemology of the word “oncogene:”

"gene which can transform a normal cell into a tumor cell," 1969, from onco- "tumor" + -gene, from root of Greek gignere (perfective genui) "to beget, produce" (from PIE root *gene- "give birth, beget"). Related: Oncogenesis "formation or production of tumors" (1932); oncogenic (1949).

The following text and screenshot are from the Nobel Prize website on Bishop and Varmus’ research in 1975 that resulted in their Nobel Prize award in 1989.

The term oncogene was introduced in the middle of the 1960s to denote special parts of the genetic material of certain viruses. It was believed that this part of the genetic material could direct the transformation of a normal cell into a tumor cell under the influence of other parts of the viral genetic material, alternatively via chemical or physical effects. The favourite theory of the time was that virus-mediated cell-to-cell transmittance of oncogenes was the origin of all forms of cancer. This view was later proven to be incorrect.

The original discovery of an oncogenic virus was made in 1916 by Peyton Rous working at the Rockefeller Institute in New York. Fifty years later Rous received the Nobel Prize in Physiology or Medicine. Rous virus, as the infectious agent later was named, is a member of a large virus family named retroviruses. The genetic material of these viruses is RNA (ribonucleic acid). This RNA can be transcribed into DNA (deoxyribonucleic acid) by a unique enzyme in the virus, reverse transcriptase. The 1975 Nobel Prize in Physiology or Medicine was awarded to David Baltimore, Renato Dulbecco and Howard Temin partly for the discovery of this enzyme.

Reverse transcription of the genetic material of the virus into DNA has the important consequence that it can become integrated into the chromosomal DNA in the cells. It was through investigations of Rous virus that this year’s laureates Michael Bishop and Harold Varmus in 1975 could demonstrate the true origin of oncogenes. They used one variant of Rous virus which contained an oncogenic gene (Figure 1) and another variant which lacked this gene. By use of these viruses they managed to construct a nucleic acid probe which selectively identified the oncogene. This probe was used to search for the corresponding genetic material in DNA from different cells. It was then found that oncogene-like material could be detected in different species throughout the animal kingdom, in fact even in simple organisms comprising only a few cells. Furthermore, it was shown that the gene had a fixed position in the chromosomes of a certain species, and that the gene, when it constituted part of the cellular genetic material, was divided into fragments (a mosaic gene) (Figure 1).

In May 1975, Robert Cardiff, of UC-Davis, published this paper on his collection of mouse mammary tumor virus (MMTV). An Abstract is all that is accessible. Robert Cardiff would soon publish a paper with Robert Malone in 1983 on the “Mouse Mammary Tumor Model.”

Long-term primary culture of mouse mammary tumor cells: production of virus, May 1975. Journal of the National Cancer Institute - Oxford. L J Young, R D Cardiff, R L Ashley

Long-term primary cultures of mouse mammary tumor cells proved an excellent source of Mouse Mammary Tumor virus (MMTV). Virus purified from these primary cultures had the same morphologic biochemical, immunologic, and biologic characteristics as MMTV.

1981. Palmiter and Brinster create brain cancer in mice using SV40.

Buoyed by their success with the MK transgenic mice, Palmiter and Brinster went on to produce transgenic mice expressing rat growth hormone, again under control of the Mt1 gene promoter (the transgene was called MGH). The result was unusually large mice (“big mice”) whose growth rates and body size were clearly enhanced by expression of the transgene-encoded hormone (Palmiter et al. 1982a, 1983). The levels of growth hormone expression from the transgene were, however, unrelated to gene copy number. In an attempt to produce higher and more reliable expression, they began testing transgene constructs that incorporated the SV40 enhancer, reasoning that the small Mt1 gene promoter fragment being used was missing an unidentified transcriptional enhancer element. For convenience, Palmiter simply fused the entire SV40 early region—including enhancer, promoter, and coding regions for large and small T-antigens—upstream to the MK or MGH genes such that the SV40 enhancer was adjacent to Mt1 gene promoter and the T-antigen genes would be transcribed in the opposite direction. These genes were called SV-MK and SV-MGH. It was generally thought at the time that mice were resistant to SV40-induced tumorigenesis, despite the fact that mouse tissue culture cells could be phenotypically transformed by T-antigen. In the summer of 1982, Brinster microinjected the SV-MK construct and generated a number of initial (founder) transgenic mice. Surprisingly, several of the founders died of unknown causes. In December, Brinster and his post-doctoral fellow Howard Chen noted that the cranium of one sickly transgenic mouse had a bulge. That mouse was sacrificed and autopsied, revealing a brain tumor (Fig. 1B). By the next summer, offspring of founder transgenic mice that carried either the SV-MK transgene, or the other hybrid gene (SV-MGH), were developing brain tumors (Supplementary Table 1).

Summer 1982. Eugenicists who are creating genetic cancer travel to Germany to further their research, naturally.

The origins of oncomice: a history of the first transgenic mice genetically engineered to develop cancer

At a meeting at the Salk Institute in summer of 1982, Wagner met Rolf Müller, who was studying the Fos oncogene at the biochemical and molecular levels. Fos was first discovered as a viral oncogene (as were Src and Myc) carried by the FBJ and FBR retroviruses, which elicited bone tumors following infection of mice. Müller and Wagner both decided to join the newly established Differentiation Program at the European Molecular Biology Laboratory (EMBL) in Heidelberg, Germany, and it seemed logical to join forces and work together to investigate the roles of c-Fos in mouse development, tissue homeostasis, and disease, by using transgenic mouse technology. A research proposal was submitted to EMBL in the fall of 1982 by Wagner and Müller, in which they proposed to work jointly on studying the roles of cellular proto-oncogenes and activated (viral) oncogenes in transgenic mice, beginning with over- and misexpressing the Fos proto-oncogene to assess its capability to act as a cancer-causing oncogene. The prospect of similarly investigating other cellular proto-oncogenes and viral oncogenes such as Src and Myc was also mentioned. In mid 1983, Wagner moved to the EMBL and focused his research efforts on the by then trendy oncogenes.

1983-1984. As an undergraduate at UC-Davis, Robert Malone is given an award to work on creating breast cancer in mice with “oncogenes.” (SV40 is an oncogene).

In 1983, Robert Malone, as an undergraduate at UC-Davis, was awarded a “President’s Undergraduate Fellowship Grant for Investigation of Oncogene Expression in Breast Tumor Tissue: 1983-1984.”

Malone’s college education began at Santa Barbara City College around 1981, where he was a teaching assistant in the computer laboratory. Malone said he changed his major from computer science to molecular biology in part because he did not want to be like his father, involved with the DoD. It looks like the cancer creation “research” opportunity for Malone played a bigger role than he has admitted.

Malone’s father went to Santa Barbara City College, as did both of his sons. Later, after gaining his knowledge on molecular biology, Malone went to the DoD. The Oncogen Expression award given to Robert Malone in 1983-1984 may have been funded under cancer development (“research”) grant program from the NIH/NIAID to UC Davis.

Robert Malone describes his mentors in creating cancer in mice with oncogenes, Murray Gardner and Bob Cardiff.

The Ripple Effect Podcast #338 (Dr. Robert W. Malone | The Inventor of mRNA Vaccines Speaks Out)

"For a variety of reasons I was motivated to be a physician and to get involved in molecular biology and molecular virology was just a passion.  And at the time breast cancer research.  So I just became a lab rat as an undergraduate in a key laboratory in the school of medicine in the department of pathology with the individuals mentoring me Murray Gardner and Bob Cardiff.   Bob was recently from the Nobel prize winning lab at UC-SF that had discovered oncogenes.   So we were very focused in that laboratory on breast cancer and the mouse mammary tumor model which is a retrovirus.”

1983-1984. Phil Leder and Tim Stewart pictured with an “oncomouse” such as Robert Malone would also have worked with. The image is from: The origins of oncomice: a history of the first transgenic mice genetically engineered to develop cancer at the Cold Springs Harbor Laboratory (CSHL).

Aside: CSHL is a Rockefeller-funded eugenics organization that transferred their research to Germany after Hitler took power, and then transferred it back to the United States with the very same German scientists after the United States defeated Germany in WWII. You can read more about Cold Springs Harbor here: Cold Springs Harbor, Eugenics, etc.

In October 1984, Robert Malone and Robert Cardiff published how they had worked with “Laboratory colonies of feral mice” that had been infected with Mouse Mammary Tumor virus (MMTV). They reported that researching an absence of some DNA presented a “unique opportunity” to create cancer in further studies.

Hyperplastic and neoplastic changes in the mammary glands of feral mice free of endogenous mouse mammary tumor virus provirus. October 1984. Journal of the National Cancer Institute - Oxford L J Faulkin, D J Mitchell, L J Young, D W Morris, R W Malone, R D Cardiff, M B Gardner

This is the only text accessible. The Abstract:

Laboratory colonies of feral mice (Mus musculus domesticus) have been established with specific mouse mammary tumor virus (MuMTV) genotype, including colonies lacking any proviral DNA (ev-) or carrying only a single copy of MuMTV DNA (ev+). No evidence of a decline in reproductive capacity has been observed in the first 8 generations. Both the ev- and ev+ mice showed normal mammary gland development and the development of hyperplastic lesions in the older females. The mice were very resistant to spontaneous or chemically induced mammary tumors. However, the occurrence of 1 mammary tumor in an ev- mouse indicates that mammary neoplasias can occur in the absence of MuMTV DNA. The few tumors that do occur in the ev- mice provide a unique opportunity to study the neoplastic process in the absence of proviral DNA.

March 1989. Robert Malone left UC Davis and the Salk Insitute and went to the startup Vical, which was backed by German pharma giant Merck, who bought the rights to all the patents of Vical. Robert Malone and Robert Cardiff, among others, applied for a Patent that listed SV40 several times.

LIPID-MEDIATED POLYNUCLEOTIDE ADMINISTRATION TO DELVER A BOLOGICALLY ACTIVE PEPTIDE AND TO INDUCE A CELLULAR IMMUNE RESPONSE

July 1997. Robert Malone is a member of the “NIH Study Section K01 Breast Cancer Study Section,” among his extensive ties as a “BioDefense” represenative to the NIH/NIAID and his Frenemy, Anthony Fauci. I’ve found no further information on the 1997 NIH Breast Cancer Study Section.

Robert Malone on Tucker Carlson, February 11, 2022.

Robert Malone descibes his background with his father, father-in-law, and the CIA. Begin 3:15.

My dad was an electrical engineer. He was deep in the Department of Defense. Particularly working in thermonuclear triggering devices and electromagnetic surge protection. High energy systems, so, a kinda spooky world. My mom was a teacher from Mills College. And really a key figure in my life was my father-in-law who was head of Raytheon's special projects, so I've met numerous people from the CIA back then who knew him.

He described his cancer “research.” He didn’t say that his research was the creation of cancer. Begin 5:02.

And went to first Santa Barbara City College.   I'm a city college grad as was my dad.  As were both of our boys. And then went to UC Davis where I really got deep into breast cancer research.   And retrovirus causes of breast cancer.   The lab I was in just serendipitously just happened to be the one that first discovered retrovirus being involved in immunodeficiency syndrome.  

After about 30 minutes of kissing his own ass, and after some prying by Tucker Carlson, Robert Malone described the massive increases in cancer, heart attacks, strokes, infertility that were reported to him by Lieutenant Colonel Theresa Long of Fort Rucker in Alabama.

Malone didn’t mention anything about SV40 or his own “research.” Instead, Malone threw Kariko and Weissman (who won the Nobel Prize in 2023) under the bus. Begin at 31:28:

The one that's popping up now is just stunning to me, is this whistleblower release of information from the DoD database. The administrative database. And in the scramble to, umm, I, fly, uhh, rewrite the data, uhh, I don't know how to say it.

Tucker: The data that describe what?

Umm, these are the data that describe the adverse events, umm, in the Department of Defense. So this. is the Defense Epidemiologic Suveillance System. And so the whistleblower... so I know Teresea Long. Speak to her fairly frequently. Spoke to her, talked to her twice this morning. Umm so she is a Lieutenant Colonel- there's three others I think, total, that are disclosing these data. So what happens is...

Tucker: I'm sorry, will you just, for our viewers who aren't following this particular story, tell us what those numbers suggest?

. .     3228. Umm,   Massive increases in 2021 relative to 2020 and preceding years of multiple hundreds of percent, up to a thousand percent, of a variety of adverse events, which is to say diseases, umm that are suspected of being associated with the vaccine administration.

Tucker: And serous adverse effects?

.   .... 33:00  Yeah, like cancer, heart attacks, stroke, uhh, infertility, it goes, uhh,... It is stunning to me, and as I've said before, I take no pleasure in finding the things I've been raising alarms about are those things that are popping up in this signal.   Ryan Cole has been observing increase incidences in cancers and odd behaviors of cancers, umm, to get in the nuance, cells replicating at a much higher level in a field of cancer cells than they would normally be seen in that type of cancer. Umm, and he's been observing this. You know, it's all anecdotal. This is just what's coming across his microscope.

Tucker: Since you ar eone. of the people in the country who understands how these vaccines work, do you think it's plausible that they increase the ...

Robert Malone. Yes.

Tucker: the risk of Cancer? You do?

Robert Malone: Yes. For, some reason, and there's just a paper out today that I cited, we were in a Substack, and also pushed out on GETTR, from an obscure journal called CELL, from an obscure University called Stanford, uhh peer-reviewed, uhh umm, that demostrates that the RNA, the synthetic RNAs, these RNAs don't have the normal chemical composition of regular RNA. The use a chemical called pseudoeurodine, which is the basis for the Kariko and Weissman claims for the Nobel Prize. And so these synthetic RNAs seem to be sticking around in the body. They only tested out for 60 days. This is not how RNA is supposed to behave. It's supposed to get degraded within hours. So for some reason these RNAs, these synthetic RNAs, are sticking around for a really long time, including in the lymph nodes. And they're yielding levels of spike protein that is higher than that that would be observed with natural infection.

END 35:20

October 2, 2023. Kariko and Weismann win the Nobel Prize. They wear masks. COVID-19 was caused by Hospitals committing homicides and falsely blaming a virus, then by the vaccines. Robert Malone conspired with Michael Callahan to create the appearance of a viral pandemic beginning in Wuhan, China.

October 19, 2023. The Epoch Times reports that Health Canada has confirmed the inclusion of SV40 DNA in the COVID-19 vaccines.

Health Canada has confirmed the presence of a Simian Virus 40 (SV40) DNA sequence in the Pfizer COVID-19 vaccine, which the manufacturer had not previously disclosed. There is debate among scientists with regards to the significance of the finding, with some saying it has the potential to cause cancer, and others saying it poses little to no threat.

October 19, 2023. Steve Kirsch calls SV40 a “contaminant.”

END

P.S. I could use some help with creating the referenced video clips. I don’t know who all can be held responsible for the inclusion of SV40, but I believe that Robert Malone is an expert on the creation of cancer with SV40 and would know exactly how to do it with a genetic “vaccine.”

REFERENCES

(1).