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What is the relationship between cancer and viruses?
Dr. Urso's and Dr. Young's comments revisited after an encouraging start to Ivermectin and Cancer community trials
On July 11, I summarized some comments of Dr. Richard Urso on hydroxychloroquine. This was before I was aware of the “Community trials” of Ivermectin and cancer. I first wrote about that international study on September 4.
I listened to Dr. Urso again today and was struck anew by what he said.
“You just proved when viruses and chloroquine are around, chloroquine will let the virus attack a cancer cell.”
Urso was referencing a study on where they used chloroquine on kidney cells and lung cells infected with SARS-2. They said chloroquine inhibited SARS in the kidney cells, but not the lung cells. There was one little catch, though, that the authors of the study hid in the appendix. The lung cells were also cancer cells.
“So I found it in the appendix, in little letters. Calu-3 lung cells. This is the disinformation campaign that we are faced with. ... It's unlikely to work against SARS-CoV-2. We just proved it. No, you just proved chloroquine is one of the smartest drugs in history. It will let viruses attack cancer cells but not normal cells.”
There’s a ton of literature out there that the best “anti-virus” medications of all time, such as Ivermectin, drugs derived from Cinchona bark (quinine, cloroquine, hydroxycloroquine), Garlic, and Artemisia also have potent anti-cancer compounds.
So what gives? What is the link between cancer and viruses?
Consider what Dr. Robert Young has repeatedly said: Viruses are repair proteins.
Under the theory of Robert Young, viruses are created by the body when it is in a state of disease. Cancer is a disease.
Continuing, these are my thoughts, these true “antiviral” compounds do not “kill” viruses; but instead bring the body back into a state of healthy balance where it stops creating viruses. When the body is in a state of disease and creating viruses to kill cancer cells, compounds like Ivermectin, hydroxycloroquine, etc., etc., do not interfere with this process, but complement it somehow.
I’m not 100% certain that I’m right. This is, in fact, a “theory.” Please discuss.
Charles Wright
What is the relationship between cancer and viruses?
The difference between the Calu and Vero cells and the results are clearly explained in the paper, and it has nothing to do with cancer. It has to do with TMPRSS2 expression, positive (+) or negative (-). Engineered TMPRSS2 (+) Vero cells showed the same ineffectiveness with Calu cells which are also TMPRSS2 (+.) Chloroquine is effective with TMPRSS2(-) Vero Cells
“Moreover, chloroquine and hydroxy- chloroquine inhibited S-driven entry into TMPRSS2− Vero cells with high efficiency whereas the inhibition of entry into TMPRSS2+ Calu-3 and TMPRSS2+ Vero cells was inefficient and absent, respectively (Fig. 1a and Table 1).
Therefore, chloroquine and hydroxychloroquine can block S-driven entry, but this inhibition is cell-line-dependent and efficient inhibition is not observed in TMPRSS2+ lung cells.”
https://www.nature.com/articles/s41586-020-2575-3.pdf
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